For research purposes, loneliness
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For research purposes, loneliness
is typically measured on a continuum
that ranges from not at all lonely (.e.,
socially connected) to very lonely. It is
informative, however, to get a sense of
the prevalence of loneliness when
assessed as present or absent. Loneliness
is a common experience; as many as 80
percent of people under 18 years of age
and 30 percent of people over 65 years
of age report being lonely at least
sometimes. For most people, feelings of
loneliness are situational and transient
(e.g., geographic relocation). For as
many as 15-30% of the general
population, however, loneliness is a
chronic state, and it is among these
individuals that loneliness wreaks its
greatest havoc. In a study of children
followed through young adulthood,
those who were highly lonely at each of
three measurement occasions (i.¢.,
childhood, adolescence, and at 26 years
of age) exhibited a significantly greater
number of standard health risks. The
chronically lonely individuals were more
likely to have higher body mass index
(BMI), elevated blood pressure, higher
levels of total cholesterol, lower levels
of “good” HDL cholesterol, greater
concentrations of glycosylated
hemoglobin (an index of impaired
glucose metabolism), and poorer
respiratory fitness than those who were
lonely at only two or one of the
measurement occasions.” In a study of
older adults, loneliness predicted
mortality over a 3-year period, and
increased mortality was explained by the
fact that lonely individuals had more
chronic diseases and functional
limitations.’ Higher rates of mortality in
lonely individuals do not appear to be
attributable to inadequate healthcare
utilization: even after accounting for the
presence and severity of chronic illness,
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lonely individuals are actually more
likely than nonlonely individuals to
make use of health facilities and
physicians.*
Most chronic diseases (e.g.,
hypertension, coronary artery disease,
diabetes) are the result of the interactive
influences of genetic, environmental,
and behavioral factors on physiological
functioning. How do feelings of
loneliness penetrate to a level that affects
disease risk? Plausible pathways include
poor health behaviors, stress-related
processes, restorative “anti-stress”
processes, and even differences in
patterns of gene activity. In general,
physiological systems exhibit
redundancies and compensatory
processes that minimize the immediate
health effects of adverse heritable,
environmental, and behavioral factors.
However, subtle changes in these
predisease pathways can be detected
prior to the onset of manifest disease and
may indicate the beginnings of a steeper
downward trajectory in resilience.”
Take health behaviors, for
instance. Major risk factors for disease
in Western society include high-calorie,
high-fat diets, and sedentary lifestyles,
each of which contribute to being
overweight or obese. Feelings of
loneliness have been associated with
greater incidence of these predominantly
lifestyle risk factors. In a large cross-
sectional survey of adults 18 years and
older, the lonely group had a higher
mean BMI and a greater proportion of
overweight/obese individuals than did
the nonlonely group. Loneliness has
been associated with lower levels of
physical activity in every age group from
grade school to middle-age adults. In the
latter study, lonely individuals were also
more likely to become inactive over
time. Changes in health status also
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